@article{oai:repository.naro.go.jp:00000734,
 author = {瀬尾, 茂美 and SEO, Shigemi and 中保, 一浩 and NAKAHO, Kazuhiro and Hong, Si Won and Hong, Si Won and 高橋, 英樹 and TAKAHASHI, Hideki and 繁森, 英幸 and SHIGEMORI, Hideyuki and 光原, 一朗 and MITSUHARA, Ichiro},
 issue = {9},
 journal = {Plant and Cell Physiology, Plant and Cell Physiology},
 month = {Sep},
 note = {Wilt disease in plants, which is caused by the soil-borne bacterial pathogen Ralstonia solanacearum, is one of the most devastating plant diseases. We previously detected bacterial wilt disease-inhibiting activity in an extract from yeast cells. In the present study, we purified this activity and identified one of the substances responsible for the activity as the amino acid histidine. The exogenous application of l-histidine, but not d-histidine, inhibited wilt disease in tomato and Arabidopsis plants without exhibiting any antibacterial activity. l-Histidine induced the expression of genes related to ethylene (ET) biosynthesis and signaling as well as the production of ET in tomato and Arabidopsis plants. l-Histidine-induced resistance to R. solanacearum was partially abolished in ein3-1, an ET-insensitive Arabidopsis mutant line. Resistance to the fungal pathogen Botrytis cinerea, which is known to require ET biosynthesis or signaling, was also induced by exogenously applied l-histidine. These results suggest that l-histidine induces resistance to R. solanacearum and B. cinerea partially through the activation of ET signaling in plants., SC10201707050024postprint-img(Supplementary data)は著者版提供より},
 pages = {1932--1942},
 title = {l-Histidine Induces Resistance in Plants to the Bacterial Pathogen                     Ralstonia solanacearum                     Partially Through the Activation of Ethylene Signaling},
 volume = {57},
 year = {2016},
 yomi = {セオ, シゲミ and ナカホ, カズヒロ and タカハシ, ヒデキ and シゲモリ, ヒデユキ and ミツハラ, イチロウ}
}